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Kta1994
Specialist
- Apr 25, 2019
- 306
Yes both seems to fit with the description. Too much possibilities to cover honestly. But one thing for sure is that both is Ischemic. Both are all about loss of oxygen to then brain. They just worded differently. It depend on the individual body type in then end. Amaurosis fugax is also a possibility. But honestly I am skeptical about one part. If its because of hypoxia that someone went temporary blind. Then that person should had fainted soon after losing visual. Because brain are really sensitive to the loss of oxygen. It doesnt make sense if hypoxia that caused the blindness. If meth b hypoxia caused the blindness it must have already triggered the cerebral hypoxia and brain would have shut down within a minute. It seems the low blood pressure hit him first before the hypoxia did. Please refer to the pics I attached.
Attachments
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Many factor into this the amount of SN you ingested, absorption rate, vit c and e in your blood. What drug you use and regimen you followed. There is no clear consensus on how long. Its quite impossible to calculate. But hypoxia is generally painless.
I see, uhm, the mechanism at play I think is common with very low pressures indeed.
Transient vision loss can occur due to a lot of things. Can't pinpoint it exactly tho
I think its most like the blood pressure. But if its blood pressure it should have been accompanied by dizziness/faintness. But yeah everyone's body is different. Their medical history. Diet and or even some underlying blood problems like that woman who went blind. Human body are just a mystery. I wonder how mine will be like?
Domperidone + Ondansetron + Mosapride would theoretically be an alternative to Meto. Meto has three distinct features: D2 and 5-HT3 antagonism, and 5-HT4 agonism.
Domperidone: D2 antagonist but less BBB passing ability than other D2 blockers, meaning less EPS and tardive dyskinesia. D2 antagonist role to prevent NV is thought to be played at CTZ located outside the BBB.
Ondansetron: 5-HT3 antagonist, which also prevent nausea and vomiting (NV) by blocking a 5-HT3 activity mainly at intestine.
Mosapride: 5-HT4 agonist, which accelerates gastric emptying, and to a lesser extent 5-HT3 antagonist. But they are available mainly in Asian countries.
I don't think there is any evidence about which antagonist (D2 or 5-HT3) has a central preventive effect on SN induced NV, so it's safe to say both antagonists are needed.
edit: D2 antagonist also acts at upper GI tract as accelerating gastric emptying.
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My understanding is that D2, 5-HT3 and HK1 antagonists (and maybe CB1 agonist and 5-HT2c antagonist) are the choices for toxic substance ingestion. I would not use mirtazapine as an antiemetic because it unfortunately seems to have none of the three antagonistic features while its strong H1 antagonism would prevent motion sickness.
Just wanted to clarify, would Food Grade SN work? That seems to be the only one I can find at the moment, but it does say 99% purity... Thoughts?
98% purity or greater is ideal. Sn NAN02 has now been proven to work, however exit international is keeping their lips sealed on more info, like like does it feel etc. Hopefully one of his next livestreams will fix this issue.
Philip Nitschke might be milking the salts attention for all its worth however.. we have the info we wanted on sn for quite some time now. Theres no reason not to say more.
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Hmm... okay thanks. I appreciate it. Guess I should get to ordering soon before stuff gets harder to get.
So these rats were caused hypoxic brain injury with SN... That sucks.
www.ncbi.nlm.nih.gov
"These findings suggest that bone marrow-derived MSCs exhibit neuroprotective effects against NaNO2-induced hypoxic brain injury through exerting anti-oxidative effects and providing energy to the brain."
" NaNO2 is highly reactive with hemoglobin causing hypoxia. "
Only fact relevant for us in this study is the fact they provoked SN brain injury btw.
www.researchgate.net
"The
brain tissue induces protective mechanisms
within minutes to limit the damage.19
However;
these protective mechanisms are lost within
hours of the hypoxic-ischemic insult."
" It is concluded that cerebellar Purkinje cells
are very vulnerable to hypoxic insult and
sodium nitrite induced hypoxia results in a
significant excitotoxic degeneration of these cells. "
So basically it seems being poisoned with SN for too many hours without dying (say you just slip to a coma) will most likely give brain damage if you don't die within the first hours due to the nitric oxide losing its neuroprotective properties, becoming damaging at one point.
Not good at all.
Bone marrow-derived mesenchymal stem cells ameliorate sodium nitrite-induced hypoxic brain injury in a rat model - PubMed
Sodium nitrite (NaNO<sub>2</sub>) is an inorganic salt used broadly in chemical industry. NaNO<sub>2</sub> is highly reactive with hemoglobin causing hypoxia. Mesenchymal stem cells (MSCs) are capable of differentiating into a variety of tissue specific cells and MSC therapy is a potential...
www.ncbi.nlm.nih.gov
" NaNO2 is highly reactive with hemoglobin causing hypoxia. "
Only fact relevant for us in this study is the fact they provoked SN brain injury btw.
(PDF) Effects of sodium nitrite-induced hypoxia on cerebellar Purkinje cells in adult rats
PDF | Objectives: To explore the changes in Purkinje cells in adult rats after inducing chemical hypoxia by sodium nitrite Methodology: Adult male albino rats, weighing 180-200 gm were used in this study. The animals were fasted for three hours prior to drugs administration. One...
www.researchgate.net
brain tissue induces protective mechanisms
within minutes to limit the damage.19
However;
these protective mechanisms are lost within
hours of the hypoxic-ischemic insult."
" It is concluded that cerebellar Purkinje cells
are very vulnerable to hypoxic insult and
sodium nitrite induced hypoxia results in a
significant excitotoxic degeneration of these cells. "
So basically it seems being poisoned with SN for too many hours without dying (say you just slip to a coma) will most likely give brain damage if you don't die within the first hours due to the nitric oxide losing its neuroprotective properties, becoming damaging at one point.
Not good at all.
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So these rats were caused hypoxic brain injury with SN... That sucks.
"These findings suggest that bone marrow-derived MSCs exhibit neuroprotective effects against NaNO2-induced hypoxic brain injury through exerting anti-oxidative effects and providing energy to the brain."Bone marrow-derived mesenchymal stem cells ameliorate sodium nitrite-induced hypoxic brain injury in a rat model - PubMed
Sodium nitrite (NaNO<sub>2</sub>) is an inorganic salt used broadly in chemical industry. NaNO<sub>2</sub> is highly reactive with hemoglobin causing hypoxia. Mesenchymal stem cells (MSCs) are capable of differentiating into a variety of tissue specific cells and MSC therapy is a potential...
" NaNO2 is highly reactive with hemoglobin causing hypoxia. "
Only fact relevant for us in this study is the fact they provoked SN brain injury btw.
"The(PDF) Effects of sodium nitrite-induced hypoxia on cerebellar Purkinje cells in adult rats
PDF | Objectives: To explore the changes in Purkinje cells in adult rats after inducing chemical hypoxia by sodium nitrite Methodology: Adult male albino rats, weighing 180-200 gm were used in this study. The animals were fasted for three hours prior to drugs administration. One...
brain tissue induces protective mechanisms
within minutes to limit the damage.19
However;
these protective mechanisms are lost within
hours of the hypoxic-ischemic insult."
" It is concluded that cerebellar Purkinje cells
are very vulnerable to hypoxic insult and
sodium nitrite induced hypoxia results in a
significant excitotoxic degeneration of these cells. "
So basically it seems being poisoned with SN for too many hours without dying (say you just slip to a coma) will most likely give brain damage if you don't die within the first hours due to the nitric oxide losing its neuroprotective properties, becoming damaging at one point.
Not good at all.
Yeah like all brain hypoxia there will be damage. After all SN kill us with it. If you gone on long enough without anyone finding you then. Yes you will get brain damage and die. If youre can still move and conscious then you wont get brain damage. If someone find you at that time. We really need to get unconscious and coma fast.
Yeah that's what scares me is that I'm found somehow when brain damage occurs. I rather use a gun for that reason...
In all honestly I myself prefer gun too. But cant buy any in my country. Gun will be so much faster and cleaner. Sigh... A single bullet. On the magnum. Aim and boom. Everything is over.
Same thought... that's how my uncle did it and how I want to as well. My country you need licence for hunting so I'd need to study and take practical lessons to then pass an exam... Something that I'm not looking forward to with my severe depression that makes me bedridden.
But for now SN is the best thing we got I guess
I am sorry to know youre bedridden. And having psychological issues practically bar us from owning a gun. Well I might do it before you so I will keep posting my experience after SN ingestion for as long as I can. Please continue to correct me if I said anything wrong. Be counting on you guys. P
I don't have any restriction to get a gun afaik actually, gov can't access medical record. Only criminal.
Sorry to hear that, but I also hope you find peace. I hang around here a lot so any thing I can help with I'll do gladly.
I see thats good to know for my country, civilian cannot own gun. Nah dont be sorry about it. I can finally be free. I can finally put an end to this. Thank you I just hope I will never wake up again. Theres nothing for me anymore in this place. Not anyone not anything.
Well I just need you guys fact check my info if is its wrong in any way. For people that came after us. The very least I want to leave them is things that can help them move on easier. No one need to suffer needlessly anymore in this world.
I will keep posting after I took SN so you guys knows what to expect. I post it in my last personal thread. Thank you for all your info
NoOneKnows
Specialist
- Sep 12, 2018
- 323
devastated. Blindness? what? I already had 2 nightmares of something similar, I woke up in fear i am blind. MY eyes condition is already bad, I have thyroid associated orbitopathy, tissues around eyes affected by autoimmune condition, does this makes me more suspectible. Good Lord..this is hell
A possibility of waking up from coma to irreversible blindness ? If there is a 0,1% chance I prefer to jump from my window, where I can end up paralyzed from waist down maybe, but still better than blindness to me ! You cant attempt ctb again when blind.
A possibility of waking up from coma to irreversible blindness ? If there is a 0,1% chance I prefer to jump from my window, where I can end up paralyzed from waist down maybe, but still better than blindness to me ! You cant attempt ctb again when blind.
Yes in some certain special cases blindness is irreversible. But its very rare and without more studies into it. We cant answer anything. But all you must do it make sure you have enough SN in your body. Dmuse meto and cime. Make surr your vit c is low. Coma long enough and youre dead. Thats the best we can do.
drakewantstogo
Pressure building
- Nov 9, 2018
- 188
On the morning I drank some milk and then ate like five sunflower seeds. Then nothing until 8pm where I drank water to help the pills go down
Raggas
Suicide is self expression
- Dec 31, 2018
- 306
I'm in Australia and it looks like Anagraine is the best option for an antiemetic (5mg metoclopramide, 500mg paracetamol - i guess 6 tablets). I can get ranitidine over the counter too. From reading through this thread, this seems feasible. But there is a lot of conflicting info, especially from the 8ch thread linked a few pages back.
Can anyone give advice?
Can anyone give advice?
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