udontknowme
New Member
- Mar 17, 2026
- 2
I know that on this forum, prescription medications overdoses are not recommended, but personally i think, having some medical knowledge can drastically increase the chances of success with CTB
I intend to take:
Xanax [4mg] 160 drops
Zolpidem [200mg] 20 tablets
Ethyl alcohol 200ml (40% alcohol by volume) (80ml of pure alcohol)
Estimated sensations I might experience:
No acute physical pain: Unlike other substances, GABA agents and alcohol induce strong sedation and sensory anaesthesia; the patient does not experience pain, but a progressive and inexorable 'shutdown' of cognitive functions.
How it works:
As early as 30–45 minutes in, the hypnotic component of zolpidem and the GABA surge from alcohol plunge the subject into a state of deep stupor, rapidly progressing to a non-responsive pharmacological coma (the patient no longer responds to verbal or painful stimuli), from which they will then silently slip into respiratory depression and hypoxia, without realising the gravity of the situation due to the shutdown of the cerebral cortex.
Prolonged respiratory depression: Even if breathing does not cease within the first minute, a chronically low respiratory rate (hypoventilation) inexorably leads to severe respiratory acidosis and progressive tissue hypoxia, which shuts down the heart and brain within a few hours.
Loss of reflexes and positional obstruction: During such a deep pharmacological coma, the neck and tongue muscles relax completely. Although the risk of vomiting is low, the tongue falling backwards (mechanical obstruction of the upper airways) is extremely likely in the supine position, leading to asphyxia within a relatively short time.
Effect with Xanax and Zolpidem: At this concentration of ethanol, the synergistic effect on GABA receptors begins to seriously compromise the protective ceiling effect of the drugs. The central nervous system enters a phase of profound depression (stupor or pre-coma). The risk of hypoventilation and the body's inability to protect the airways (risk of choking on vomit) becomes very high.
Expected clinical picture: Deep or prolonged coma, severe bradypnoea (respiratory rate 8 breaths per minute), cyanosis, cardiovascular collapse and an extremely high risk of respiratory arrest or death due to central bulbar obstruction/depression, requiring immediate mechanical ventilation and intensive care.
Estimated success rates:
First hour:
Symptoms: Initial deep coma, breathing still rhythmic but slowed, possible slight drooling or muscle relaxation.
Fatalities (without medical assistance): < 5 per cent.
Probability of success (with prompt medical assistance): > 95 per cent (recovery almost certain).
Second - Third hour:
Symptoms: Marked bradypnoea, completely flaccid musculature, onset of slightly stertorous breathing due to fluoxetine's inhibition of drug metabolism.
Mortality (without treatment): 20–40 per cent.
Probability of success (with treatment): 80–90 per cent (high efficacy of mechanical ventilation).
Third - Fifth hour:
Symptoms: Deep stertor, anoxic rales, obvious respiratory pauses (apnoea) alternating with gasps or desperate attempts at inspiration
Mortality (without medical assistance): 75–95 per cent.
Probability of survival (with medical assistance): 30–50 per cent (high risk of permanent brain damage due to prolonged hypoxia).
Fifth - Seventh hour:
Patient's symptoms: Complete respiratory silence (terminal apnoea), marked cyanosis of the lips and fingers, body completely limp and cold.
Mortality (without medical assistance): > 95%.
Probability of success (with medical assistance): < 15% (full-blown anoxic cardiac arrest; resuscitation often futile).
Beyond the eighth hour:
Patient's symptoms: Death has occurred; appearance of post-mortem signs such as rigor mortis or lividity.
Fatal outcome (without medical assistance): 100%.
Probability of survival (with resuscitation): 0% (incompatible with life).
I'd like to know what you think, particularly if there's anyone here studying clinical pharmacology, neuroscience, toxicology, or who otherwise has a grounding and expertise in the subject
But in any case, any comments are welcome
(I hope my English is understandable; it's not my first language, lol)
I intend to take:
Xanax [4mg] 160 drops
Zolpidem [200mg] 20 tablets
Ethyl alcohol 200ml (40% alcohol by volume) (80ml of pure alcohol)
Estimated sensations I might experience:
No acute physical pain: Unlike other substances, GABA agents and alcohol induce strong sedation and sensory anaesthesia; the patient does not experience pain, but a progressive and inexorable 'shutdown' of cognitive functions.
How it works:
As early as 30–45 minutes in, the hypnotic component of zolpidem and the GABA surge from alcohol plunge the subject into a state of deep stupor, rapidly progressing to a non-responsive pharmacological coma (the patient no longer responds to verbal or painful stimuli), from which they will then silently slip into respiratory depression and hypoxia, without realising the gravity of the situation due to the shutdown of the cerebral cortex.
Prolonged respiratory depression: Even if breathing does not cease within the first minute, a chronically low respiratory rate (hypoventilation) inexorably leads to severe respiratory acidosis and progressive tissue hypoxia, which shuts down the heart and brain within a few hours.
Loss of reflexes and positional obstruction: During such a deep pharmacological coma, the neck and tongue muscles relax completely. Although the risk of vomiting is low, the tongue falling backwards (mechanical obstruction of the upper airways) is extremely likely in the supine position, leading to asphyxia within a relatively short time.
Effect with Xanax and Zolpidem: At this concentration of ethanol, the synergistic effect on GABA receptors begins to seriously compromise the protective ceiling effect of the drugs. The central nervous system enters a phase of profound depression (stupor or pre-coma). The risk of hypoventilation and the body's inability to protect the airways (risk of choking on vomit) becomes very high.
Expected clinical picture: Deep or prolonged coma, severe bradypnoea (respiratory rate 8 breaths per minute), cyanosis, cardiovascular collapse and an extremely high risk of respiratory arrest or death due to central bulbar obstruction/depression, requiring immediate mechanical ventilation and intensive care.
Estimated success rates:
First hour:
Symptoms: Initial deep coma, breathing still rhythmic but slowed, possible slight drooling or muscle relaxation.
Fatalities (without medical assistance): < 5 per cent.
Probability of success (with prompt medical assistance): > 95 per cent (recovery almost certain).
Second - Third hour:
Symptoms: Marked bradypnoea, completely flaccid musculature, onset of slightly stertorous breathing due to fluoxetine's inhibition of drug metabolism.
Mortality (without treatment): 20–40 per cent.
Probability of success (with treatment): 80–90 per cent (high efficacy of mechanical ventilation).
Third - Fifth hour:
Symptoms: Deep stertor, anoxic rales, obvious respiratory pauses (apnoea) alternating with gasps or desperate attempts at inspiration
Mortality (without medical assistance): 75–95 per cent.
Probability of survival (with medical assistance): 30–50 per cent (high risk of permanent brain damage due to prolonged hypoxia).
Fifth - Seventh hour:
Patient's symptoms: Complete respiratory silence (terminal apnoea), marked cyanosis of the lips and fingers, body completely limp and cold.
Mortality (without medical assistance): > 95%.
Probability of success (with medical assistance): < 15% (full-blown anoxic cardiac arrest; resuscitation often futile).
Beyond the eighth hour:
Patient's symptoms: Death has occurred; appearance of post-mortem signs such as rigor mortis or lividity.
Fatal outcome (without medical assistance): 100%.
Probability of survival (with resuscitation): 0% (incompatible with life).
I'd like to know what you think, particularly if there's anyone here studying clinical pharmacology, neuroscience, toxicology, or who otherwise has a grounding and expertise in the subject
But in any case, any comments are welcome
(I hope my English is understandable; it's not my first language, lol)
