Resource SN - A Comprehensive guide including method

[Stan]

I don't know why it should be ruined...ppl with try to ask questions that are not in the main topic...the difference between this and the previous one is the previous don't have so much useful info on the first page so people had to scroll 180 pages to get what they wanted and maybe even after that they weren't able to find the asnwer so I thought ppl will ask questions there and you can add it to the FAQ so next time ppl will find it there right on the first page

Where in the land of all things holy do you think my research stretched to a baldness cure/prevention whilst cbt?

 

[a_strange_day]

haha ! The next one should be a megathread to help identify all SN threads slowly becoming megathreads talking about the SN megathread. Should be fun.

 

[MeltingHeart]

haha ! The next one should be a megathread to help identify all SN threads slowly becoming megathreads talking about the SN megathread. Should be fun.

could be called SN- sanctioned nonsensical's or perhaps - Standardised Non sequitur's?

 

[fightingsioux]

I don't know why it should be ruined...ppl with try to ask questions that are not in the main topic...the difference between this and the previous one is the previous don't have so much useful info on the first page so people had to scroll 180 pages to get what they wanted and maybe even after that they weren't able to find the asnwer so I thought ppl will ask questions there and you can add it to the FAQ so next time ppl will find it there right on the first page

With all due respect, I think you're missing the point here.

He has obviously given the project an incredible amount of thought and effort and spent many, many hours over many, many days creating a document that answers every single REASONABLE question about the subject.

Within an hour posters were asking questions that were already answered in the document.

 

[nooo2]

This is a blessing, thank you so much Stan.

 

[*KNAZ*]

I see your point but the peak concentration of Domperidone is 1 hour after you take it. So once the acid reducer is in your stomach it will begin effecting the absorption of Domperidone. That's why they put the 2 hour guideline because it will reduce bioavailability and the overall absorption of Domperidone. That means less of a dose gets to your bloodstream and higher chance of vomiting.

Is the best option to skip the acid reducer or does it really have a vital role to play in making SN method effective? I can't tolerate meto so I'm kinda stuck here.

You are making my eyes bleed

 

[FallenX]

You are making my eyes bleed

You don't understand how serious it is to take antacids or acid reducers with Domperidone. It pretty much makes the medication ineffective. Then you puke and you're screwed

 

[*KNAZ*]

You don't understand how serious it is to take antacids or acid reducers with Domperidone. It pretty much makes the medication ineffective. Then you puke and you're screwed

I'm going to be drinking 25 g of SN. Anything else is insignificant to me.

 

[BPD_LE]

Well....... I salute your attempt @Stan my friend. Let's have a popcorn fight!

 

[Haku]

Stan, you are incredible in putting this thread together, I think you've answered every question that I have, thank you. will be finishing off my preparations tonight.

 

[Heart of Ice]

Sorry if this has been said before, but taking Ibuprofen on an empty stomach is a bad idea. Thanks for the guide, it will help people.

 

[Stan]

Sorry if this has been said before, but taking Ibuprofen on an empty stomach is a bad idea. Thatns for the guide, it will help people.

only if you make a habit of it over a period of weeks as it could give you stomach ulcers. A one off dose will not hurt!

 

[Heart of Ice]

only if you make a habit of it over a period of weeks as it could give you stomach ulcers. A one off dose will not hurt!

Ibuprofen might give you a stomach ache if you take it on an empty stomach. It's nothing major, but this has happened to me. Maybe I'm just sensitive to it, I don't know.

The Ibuprofen I have specifically mentions that you should not take it on an empty stomach. Milk helps.

 

[CaptainT]

Stan's guide is a big update to previous threads and the PPHB because it's eliminating one of the three classic drugs in the SN cocktail (strong anti-acid)

Are we confident that all that's needed is 1) SN and 2) The antiemetic ?

For people who have to replace metoclopramide with domperidone the anti-acid (even an otc one) is not an option anyway so curious what others in this situation recommend?

 

[Stan]

So thanks to those who have said nice things regarding the original post, means a lot. Thanks to those who help to make the next version clearer and more understandable, over the next few days I will make a new updated first post with those inclusions/changes in there and ask the mods to delete this one eventually as not to confuse people with two versions in flight at the same time.

So let me explain how I came to the op. The PPH (specifically in the SN method to me is seemingly designed for those with physical ailments and are not on a cocktail of drugs to assist with their MH issues. A lot of posts here show quite rightly individuals looking at the regimen and highlight concerns with regard to adverse effects with their ongoing treatment. Especially in the areas of meto and acid regulators. In the past I came up with a crazy idea or two on how to get the SN into your system, and then my further research discounted them. So this is the approach I took to how the oral intake of SN could be done specifically for that profile of person. I got a clean piece of paper and started to track (with 100% help from the internet) the journey of SN from the mouth to the point SN entered the blood stream and all the metabolic processes that the body employs to remove toxins. Also the effect that SN has on our physiology to see if anything can enhance the process in either speed or efficiency. Doing that, I then cross referenced the PPH suggested regimen to understand what component works for what part of the digestive process. Hopefully some people would have noticed that through my research I said that SN is not a practical solution for those that suffer ulcers anywhere in their digestive tract based on this research. That is not in the PPH!

So I am going to go through the 'Sesame Street' version of human digestion so you get closer to why I suggested the above. So you drink the SN, it hits your mouth, nothing there to be seen or done for digestion. Goes down the gullet, meets a sphincter that controls both the entry of food into the stomach and stops gastric juices coming up which will give pain if allowed to do so. Then it hits the stomach. The stomach itself does nothing to send nutrients into the bloodstream. Its a big grinding machine that infuses the 'food' with enzymes and acid to help break down the food into a mush before the sphincter at the bottom end of your stomach allows passage into the small intestine.

Ok - so what is this gastric juices? Well there are a lot of enzymes and this thing called Hydrochloric Acid. A chemical! Interesting! What does that mean? Chemical formula for SN meeting your gastric juices would be 3NaNO2 + 2HCl → 2NaCl + NaNO3 + 2NO + H2O. Still lost me! Can you speak English please? So basically, when SN hits your stomach, the acid there works to convert it into other things such as regular table salt, Nitric oxide, Sodium Nitrate and water. None of those are really what you want. So reducing the HCL prevents SN being converted to something else? Yes, correct. Well how do we minimise this? It needs something to neutralise the HCL acid. More commonly you want to create a chemical reaction like this, Mg(OH)2(s) + 2HCl(aq) → 2H2O(l) + MgCl2(aq). You are at it again Stan! Speak English damn you! OK - what you need is an antacid with magnesium hydroxide in it. Why do you recommend that one over others? Well there are a number of other alkali that neutralise stomach acid, but this is the most benign on the list and has minimal to no conflict with other medications. It is generally known as Milk of Magnesia. Where do you find that? Any regular pharmacy without prescription. Can I use other non prescription antacid? Yes you can, I just stated Milk of Magnesia as it was the easiest when looking at some other drug interactions. But most are simple and cause no adverse effects.

So the SN is in our stomach, but you said that's not the place where the SN moves into the bloodstream. That's correct! Two main sphincters in the stomach, the one at the top already addressed and the one at the bottom which controls the movement into the small intestine where the nutrients (or SN in this case) transfers (and I am over simplifying this) into the bloodstream. Ah! So using meto is vital because it moves things faster into the small intestine? No! If you think that the reason Meto exists is for people who have conditions such as Gastroparesis where they cannot pass food very well. The key word here is food. What we are doing if we have followed the fasting process is give the stomach water. Yes, the SN is suspended in water but the stomach isn't a complex lab where it tests everything before it passes it through. But it does recognises two things. One that it is water and the body craves hydration so it will give that substance a priority pass to move to the small intestine. Secondly, the stomach does not need to mash it up to make it easier for the small intestine to cope with.

So what does that mean in conclusion? This is up for your debate and I take no offence in challenges, rebukes or proof that it is wrong. After all, this is a resource to help everyone pass peacefully with minimum stress and discomfort. Remember my premise for investigating this was that people understood that some of the regimen drugs recommended where in conflict with their existing prescription. Plus my personal opinion that the PPH has over-baked the regimen to reach the end goal (preparing yourself physically to ingest the SN).

Regimen Drug	Objective of Drug	Alternative view
Metoclopramide	antiemetic Moves food quicker into small intestine	If you have followed the fast. The natural human digestive process will move the liquid quickly into the small intestine to help the body hydrate. With this fact out of the way, you could then look for an antiemetic that does not conflict with your existing meds (as long as it it the right type)
Ranitidine	Reduce stomach acid over a period of time	A drug that will reduce stomach acid but may also have serious interactions with other meds. The goal is to reduce the HCL in the stomach and the same goal can be reached by taking a stat dose of Milk of magnesia.

 

[Ln42]

So thanks to those who have said nice things regarding the original post, means a lot. Thanks to those who help to make the next version clearer and more understandable, over the next few days I will make a new updated first post with those inclusions/changes in there and ask the mods to delete this one eventually as not to confuse people with two versions in flight at the same time.

So let me explain how I came to the op. The PPH (specifically in the SN method to me is seemingly designed for those with physical ailments and are not on a cocktail of drugs to assist with their MH issues. A lot of posts here show quite rightly individuals looking at the regimen and highlight concerns with regard to adverse effects with their ongoing treatment. Especially in the areas of meto and acid regulators. In the past I came up with a crazy idea or two on how to get the SN into your system, and then my further research discounted them. So this is the approach I took to how the oral intake of SN could be done specifically for that profile of person. I got a clean piece of paper and started to track (with 100% help from the internet) the journey of SN from the mouth to the point SN entered the blood stream and all the metabolic processes that the body employs to remove toxins. Also the effect that SN has on our physiology to see if anything can enhance the process in either speed or efficiency. Doing that, I then cross referenced the PPH suggested regimen to understand what component works for what part of the digestive process. Hopefully some people would have noticed that through my research I said that SN is not a practical solution for those that suffer ulcers anywhere in their digestive tract based on this research. That is not in the PPH!

So I am going to go through the 'Sesame Street' version of human digestion so you get closer to why I suggested the above. So you drink the SN, it hits your mouth, nothing there to be seen or done for digestion. Goes down the gullet, meets a sphincter that controls both the entry of food into the stomach and stops gastric juices coming up which will give pain if allowed to do so. Then it hits the stomach. The stomach itself does nothing to send nutrients into the bloodstream. Its a big grinding machine that infuses the 'food' with enzymes and acid to help break down the food into a mush before the sphincter at the bottom end of your stomach allows passage into the small intestine.

Ok - so what is this gastric juices? Well there are a lot of enzymes and this thing called Hydrochloric Acid. A chemical! Interesting! What does that mean? Chemical formula for SN meeting your gastric juices would be 3NaNO2 + 2HCl → 2NaCl + NaNO3 + 2NO + H2O. Still lost me! Can you speak English please? So basically, when SN hits your stomach, the acid there works to convert it into other things such as regular table salt, Nitric oxide, Sodium Nitrate and water. None of those are really what you want. So basically, reducing the HCL prevents SN being converted to something else? Yes, correct. Well how do we minimise this? Basically it needs something to neutralise the HCL acid. More commonly you want to create a chemical reaction like this, Mg(OH)2(s) + 2HCl(aq) → 2H2O(l) + MgCl2(aq). You are at it again Stan! Speak English damn you! OK - what you need is an antacid with magnesium hydroxide in it. Why do you recommend that one over others? Well there are a number of other alkali that neutralise stomach acid, but this is the most benign on the list and has minimal to no conflict with other medications. It is generally known as Milk of Magnesia. Where do you find that? Any regular pharmacy without prescription.

So the SN is in our stomach, but you said that's not the place where the SN moves into the bloodstream. That's correct! Two main sphincters in the stomach, the one at the top already addressed and the one at the bottom which controls the movement into the small intestine where the nutrients (or SN in this case) transfers (and I am over simplifying this) into the bloodstream. Ah! So using meto is vital because it moves things faster into the small intestine? No! If you think that the reason Meto exists is for people who have conditions such as Gastroparesis where they cannot pass food very well. The key word here is food. What we are doing if we have followed the fasting process is give the stomach water. Yes, the SN is suspended in water but the stomach isn't a complex lab where it tests everything before it passes it through. But it does recognises two things. One that it is water and the body craves hydration so it will give that substance a priority pass to move to the small intestine. Secondly, the stomach does not need to mash it up to make it easier for the small intestine to cope with.

So what does that mean in conclusion? This is up for your debate and I take no offence in challenges, rebukes or proof that it is wrong. After all, this is a resource to help everyone pass peacefully with minimum stress and discomfort. Remember my premise for investigating this was that people understood that some of the regimen drugs recommended where in conflict with their existing prescription. Plus my personal opinion that the PPH has over-baked the regimen to reach the end goal (preparing yourself physically to ingest the SN).

Regimen Drug	Objective of Drug	Alternative view
Metoclopramide	antiemetic Moves food quicker into small intestine	If you have followed the fast. The natural human digestive process will move the liquid quickly into the small intestine to help the body hydrate. With this fact out of the way, you could then look for an antiemetic that does not conflict with your existing meds (as long as it it the right type)
Ranitidine	Reduce stomach acid over a period of time	A drug that will reduce stomach acid but may also have serious interactions with other meds. The goal is to reduce the HCL in the stomach and the same goal can be reached by taking a stat dose of Milk of magnesia.

Great work! Thank you for helping me understand the process better and thank you for your hard work, it is noticed and appreciated.

Is there any chance you could include the recommended doses of propranolol and or benzo's, if you have these? I know they are optional extra's but I can't find where the pph said the recommended dose of propranolol. I understand if this is something you don't want to add but I thought I would ask.

Thanks again Stan.

 

[Roger]

Milk of Magnesia - is this the first mention of it ? Is it an "either/or" ingredient vis à vis Ranitidine?

MoM has a laxative effect. We go to great lengths to use the proper antiemetic to prevent ejection of SN via the mouth, but is there any danger of loss of SN from the other end ?

 

[Daniela]

MoM has a laxative effect. We go to great lengths to use the proper antiemetic to prevent ejection of SN via the mouth, but is there any danger of loss of SN from the other end ?

To be fair, by the time you pass stool some of it HAS to be in your system.

 

[FallenX]

I bought Zantac, then researched what the antacid was actually for and realised myself it was overkill for the job. Sometimes the PPH go too overboard on the regimen drugs in my opinion and having these potential negative drug interactions causes (quite rightfully) concern.

In your opinion a person can still be very successful without the acid reducer? Like reliability of SN won't decrease?

Sucks I can't take meto. I hope enough SN can enter my bloodstream with just fasting and no acid reducer.

 

[Stan]

In your opinion a person can still be very successful without the acid reducer? Like reliability of SN won't decrease?

Sucks I can't take meto. I hope enough SN can enter my bloodstream with just fasting and no acid reducer.

have you read what I have written?

 

[BPD_LE]

Obviously not. Popcorn?

 

[Stan]

Someone type "what type of water do I need?" I double dare you?

 

[FallenX]

I bought Zantac, then researched what the antacid was actually for and realised myself it was overkill for the job. Sometimes the PPH go too overboard on the regimen drugs in my opinion and having these potential negative drug interactions causes (quite rightfully) concern.

Yeah I read what you wrote and that you recommended milk of magnesia instead. Which still reduces stomach acid so it's contraindicated with Domperidone.

Just basing off the OP that says stomach acid turns SN into regular salt. If that's true are my chances of being successful much less since I'm not taking the acid reducer?

If I take the reducer I may vomit cause my anti emetic isn't being absorbed properly

If I don't take it less of the amount of SN is absorbed cause of stomach acid

I remember your post "if I had a choice I would skip the acid reducer". So it won't make a big difference on how much SN is absorbed or on if I get a lethal dose in my system after passing through the stomach?

 

[BPD_LE]

 

[hobbydevil]

This thread has been very informative. Thanks for that, Stan.

It has also been incredibly entertaining.

 

[Stan]

My reaction to the question "will this interfere with my baldness treatment?"

 

[Daniela]

Question: @Stan, were you here when Shawn Shatto did it? Remember anything, have - perchance - a single screenshot? Did she deviate from the protocol?

 

[Stan]

Question: @Stan, were you here when Shawn Shatto did it? Remember anything, have - perchance - a single screenshot? Did she deviate from the protocol?

what's that got to do with the point of this thread?

 

[Daniela]

what's that got to do with the point of this thread?

Because she CTB'd and I want to know how a person who attempted in circumstances similar to mine succeeded

 

[Stan]