HannahB
Death is the true name of time.
- Oct 29, 2019
- 187
I see many people with lack of access to the popular methods of ctb so I want to offer 'free' natural methods of ctb.
1) Water Hemlock
Closely related to poison hemlock (the plant that famously killed Socrates), water hemlock has been deemed "the most violently toxic plant in North America." A large wildflower in the carrot family, water hemlock resembles Queen Anne's lace and is sometimes confused with edible parsnips or celery. However, water hemlock is infused with deadly cicutoxin, especially in its roots, and will rapidly generate potentially fatal symptoms in anyone unlucky enough to eat it. Painful convulsions, abdominal cramps, nausea, and death are common, and those who survive are often afflicted with amnesia or lasting tremors.
Cicuta, commonly known as water hemlock, is a genus of four species of highly poisonous plants in the family Apiaceae. They are perennial herbaceousplants which grow up to 2.5 meters (8.2 ft) tall, having distinctive small green or white flowers arranged in an umbrella shape (umbel). Plants in this genus may also be referred to as cowbane or poison parsnip. Cicuta is native to temperate regions of the Northern Hemisphere, mainly North America and Europe, typically growing in wet meadows, along streambanks and other wet and marshy areas. These plants bear a close resemblance to other members in the family Apiaceae and may be confused with a number of other edible and poisonous plants. The common name hemlock may also be confused with poison hemlock (Conium maculatum), or with the Hemlock tree.
Deadly Nightshade
According to legend, Macbeth's soldiers poisoned the invading Danes with wine made from the sweet fruit of deadly nightshade. Indeed, it is the sweetness of the berries that often lures children and unwitting adults to consume this lethal plant. A native of wooded or waste areas in central and southern Eurasia, deadly nightshade has dull green leaves and shiny black berries about the size of cherries. Nightshade contains atropine and scopolamine in its stems, leaves, berries, and roots, and causes paralysis in the involuntary muscles of the body, including the heart. Even physical contact with the leaves may cause skin irritation.
Atropa belladonna, commonly known as belladonnaor deadly nightshade, is a perennial herbaceous plantin the nightshade family Solanaceae, which includes tomatoes, potatoes, and eggplant (aubergine). It is native to Europe, North Africa, and Western Asia. Its distribution extends from Great Britain in the west to western Ukraine and the Iranian province of Gilan in the east. It is also naturalised or introduced in some parts of Canada and the United States.
The foliage and berries are extremely toxic when ingested, containing tropane alkaloids.[1][2] These toxins include atropine, scopolamine and hyoscyamine, which cause delirium and hallucinations,[1][2][3] and are also used as pharmaceutical anticholinergics. These tropane alkaloids appear to be common in the family Solanaceae, as they are also present in plants of the genera Brugmansia, Datura and Hyoscyamus, of the same family but in different subfamilies and tribes than the nightshade.
Atropa belladonna has unpredictable effects.[1] The antidote for belladonna poisoning is physostigmineor pilocarpine, the same as for atropine.[4]
White Snakeroot
An innocuous plant, white snakeroot was responsible for the death of Abraham Lincoln's mother, Nancy Hanks. White snakeroot is a North American herb with flat-topped clusters of small white flowers and contains a toxic alcohol known as trematol. Unlike those who have died from directly ingesting deadly plants, poor Nancy Hanks was poisoned by simply drinking the milk of a cow who had grazed on the plant. Indeed, both the meat and milk from poisoned livestock can pass the toxin to human consumers. Symptoms of "milk poisoning" include loss of appetite, nausea, weakness, abdominal discomfort, reddened tongue, abnormal acidity of the blood, and death. Luckily farmers are now aware of this life-threatening hazard and make efforts remove the plant from animal pastures.
Ageratina altissima, also known as white snakeroot,[2]richweed,[2] or white sanicle,[3] is a poisonous perennial herb in the family Asteraceae, native to eastern and central North America. An older binomial name for this species is Eupatorium rugosum, but the genus Eupatorium has undergone taxonomic revision by botanists, and a number of the species that were once included in it have been moved to other genera.
Plants are upright or sometimes ascending, growing to 1.5 metres (4.9 ft) tall, producing single or multi-stemmed clumps in mid to late summer and fall. The flowers are a clean white color and after blooming, small seeds with fluffy white tails are released to blow in the wind. They are found in woods and brush thickets where they bloom mid to late summer or fall. The species is adaptive to different growing conditions; it is found in woods and brush thickets and also in shady areas with open bare ground, and can be weedy in shady landscapes and hedgerows. There are two different varieties: Ageratina altissimavar. altissima and Ageratina altissima var. roanensis(Appalachian white snakeroot); they differ in the length of the flower phyllaries and shape of the apices.[4][5]
White snakeroot contains the toxin tremetol; when the plants are consumed by cattle, the meat and milk become contaminated with the toxin. When milk or meat containing the toxin is consumed, the poison is passed on to humans. If consumed in large enough quantities, it can cause tremetol poisoning in humans. The poisoning is also called milk sickness, as humans often ingested the toxin by drinking the milk of cows that had eaten snakeroot.[6]
Rosary Pea
Also called jequirity beans, these piously-named seeds contain abrin, an extremely deadly ribosome-inhibiting protein. Rosary peas are native to tropical areas and are often used in jewelry and prayer rosaries. While the seeds are not poisonous if intact, seeds that are scratched, broken, or chewed can be lethal. It only takes 3 micrograms of abrin to kill an adult, less than the amount of poison in one seed, and it is said that numerous jewelry makers have been made ill or died after accidentally pricking their fingers while working with the seeds. Like ricin, abrin prevents protein synthesis within cells and can cause organ failure within four days.
Abrus precatorius, commonly known as jequirity bean or rosary pea, is a herbaceous flowering plant in the bean family Fabaceae. It is a slender, perennial climber with long, pinnate-leafleted leaves that twines around trees, shrubs, and hedges.
The toxin abrin is a dimer consisting of two proteinsubunits, termed A and B. The B chain facilitates abrin's entry into a cell by bonding to certain transport proteins on cell membranes, which then transport the toxin into the cell. Once inside the cell, the A chain prevents protein synthesis by inactivating the 26S subunit of the ribosome. One molecule of abrin will inactivate up to 1,500 ribosomes per second.
Symptoms are identical to those of ricin, except abrinis more toxic by almost two orders of magnitude; the fatal dose of abrin is approximately 1/75th that of the fatal dose of ricin (though the abrin in ingested seeds may be absorbed much more slowly than the ricin in Ricinus communis even if the seeds are chewed and the coat penetrated, allowing time for successful rescue efforts in at least some cases.[8]) Abrin has an LD50 of only 0.56 μg/kg in mice, and Kingsbury lists a toxic dose in humans at 0.00015% body weight, or approximately 0.1 mg for a 150 lb human.[9] Ingesting intact seeds may result in no clinical findings, as they can pass undigested through the gastrointestinal tract because of their hard shell.[10]
This plant is also poisonous to horses.[11]
Symptoms of poisoning include nausea, vomiting, convulsions, liver failure, and death, usually after several days.
Death Cap
Perhaps the deadliest of all mushrooms, the death cap is found throughout Europe and closely resembles edible straw mushrooms and caesar's mushrooms. Its heat-stable amatoxins withstand cooking temperatures and quickly damage cells throughout the body. Within 6 to 12 hours after consumption, violent abdominal pain, vomiting, and bloody diarrhea appear, causing rapid loss of fluid from the tissues and intense thirst. Signs of severe involvement of the liver, kidneys, and central nervous system soon follow, including a decrease in urinary output and a lowering of blood sugar. This condition leads to coma and death in more than 50 percent of the incidents. Notable deaths include Pope Clement VII, who died of accidental death cap poisoning in 1534, and possibly Roman Emperor Claudius in 54 CE.
Amanita phalloides /æməˈnaɪtə fəˈlɔɪdiːz/, commonly known as the death cap, is a deadly poisonous basidiomycete fungus, one of many in the genus Amanita. Widely distributed across Europe, but now sprouting in other parts of the world, A. phalloidesforms ectomycorrhizas with various broadleaved trees. In some cases, the death cap has been introduced to new regions with the cultivation of non-native species of oak, chestnut, and pine. The large fruiting bodies (mushrooms) appear in summer and autumn; the caps are generally greenish in colour with a white stipe and gills. Cap colour is variable, including white forms (see Taxonomy below), and thus not a reliable identifier.
These toxic mushrooms resemble several edible species (most notably Caesar's mushroom and the straw mushroom) commonly consumed by humans, increasing the risk of accidental poisoning. Amatoxins, the class of toxins found in these mushrooms, are thermostable: they resist changes due to heat, so their toxic effects are not reduced by cooking.
A. phalloides is one of the most poisonous of all known mushrooms. It is estimated that as little as half a mushroom contains enough toxin to kill an adult human. It has been involved in the majority of human deaths from mushroom poisoning,[1] possibly including the deaths of Roman Emperor Claudius in AD 54 and Holy Roman Emperor Charles VI in 1740. It has been the subject of much research, and many of its biologically active agents have been isolated. The principal toxic constituent is α-amanitin, which damages the liver and kidneys, causing liver and kidney failure that can be fatal
1) Water Hemlock
Closely related to poison hemlock (the plant that famously killed Socrates), water hemlock has been deemed "the most violently toxic plant in North America." A large wildflower in the carrot family, water hemlock resembles Queen Anne's lace and is sometimes confused with edible parsnips or celery. However, water hemlock is infused with deadly cicutoxin, especially in its roots, and will rapidly generate potentially fatal symptoms in anyone unlucky enough to eat it. Painful convulsions, abdominal cramps, nausea, and death are common, and those who survive are often afflicted with amnesia or lasting tremors.
Cicuta, commonly known as water hemlock, is a genus of four species of highly poisonous plants in the family Apiaceae. They are perennial herbaceousplants which grow up to 2.5 meters (8.2 ft) tall, having distinctive small green or white flowers arranged in an umbrella shape (umbel). Plants in this genus may also be referred to as cowbane or poison parsnip. Cicuta is native to temperate regions of the Northern Hemisphere, mainly North America and Europe, typically growing in wet meadows, along streambanks and other wet and marshy areas. These plants bear a close resemblance to other members in the family Apiaceae and may be confused with a number of other edible and poisonous plants. The common name hemlock may also be confused with poison hemlock (Conium maculatum), or with the Hemlock tree.
Deadly Nightshade
According to legend, Macbeth's soldiers poisoned the invading Danes with wine made from the sweet fruit of deadly nightshade. Indeed, it is the sweetness of the berries that often lures children and unwitting adults to consume this lethal plant. A native of wooded or waste areas in central and southern Eurasia, deadly nightshade has dull green leaves and shiny black berries about the size of cherries. Nightshade contains atropine and scopolamine in its stems, leaves, berries, and roots, and causes paralysis in the involuntary muscles of the body, including the heart. Even physical contact with the leaves may cause skin irritation.
Atropa belladonna, commonly known as belladonnaor deadly nightshade, is a perennial herbaceous plantin the nightshade family Solanaceae, which includes tomatoes, potatoes, and eggplant (aubergine). It is native to Europe, North Africa, and Western Asia. Its distribution extends from Great Britain in the west to western Ukraine and the Iranian province of Gilan in the east. It is also naturalised or introduced in some parts of Canada and the United States.
The foliage and berries are extremely toxic when ingested, containing tropane alkaloids.[1][2] These toxins include atropine, scopolamine and hyoscyamine, which cause delirium and hallucinations,[1][2][3] and are also used as pharmaceutical anticholinergics. These tropane alkaloids appear to be common in the family Solanaceae, as they are also present in plants of the genera Brugmansia, Datura and Hyoscyamus, of the same family but in different subfamilies and tribes than the nightshade.
Atropa belladonna has unpredictable effects.[1] The antidote for belladonna poisoning is physostigmineor pilocarpine, the same as for atropine.[4]
White Snakeroot
An innocuous plant, white snakeroot was responsible for the death of Abraham Lincoln's mother, Nancy Hanks. White snakeroot is a North American herb with flat-topped clusters of small white flowers and contains a toxic alcohol known as trematol. Unlike those who have died from directly ingesting deadly plants, poor Nancy Hanks was poisoned by simply drinking the milk of a cow who had grazed on the plant. Indeed, both the meat and milk from poisoned livestock can pass the toxin to human consumers. Symptoms of "milk poisoning" include loss of appetite, nausea, weakness, abdominal discomfort, reddened tongue, abnormal acidity of the blood, and death. Luckily farmers are now aware of this life-threatening hazard and make efforts remove the plant from animal pastures.
Ageratina altissima, also known as white snakeroot,[2]richweed,[2] or white sanicle,[3] is a poisonous perennial herb in the family Asteraceae, native to eastern and central North America. An older binomial name for this species is Eupatorium rugosum, but the genus Eupatorium has undergone taxonomic revision by botanists, and a number of the species that were once included in it have been moved to other genera.
Plants are upright or sometimes ascending, growing to 1.5 metres (4.9 ft) tall, producing single or multi-stemmed clumps in mid to late summer and fall. The flowers are a clean white color and after blooming, small seeds with fluffy white tails are released to blow in the wind. They are found in woods and brush thickets where they bloom mid to late summer or fall. The species is adaptive to different growing conditions; it is found in woods and brush thickets and also in shady areas with open bare ground, and can be weedy in shady landscapes and hedgerows. There are two different varieties: Ageratina altissimavar. altissima and Ageratina altissima var. roanensis(Appalachian white snakeroot); they differ in the length of the flower phyllaries and shape of the apices.[4][5]
White snakeroot contains the toxin tremetol; when the plants are consumed by cattle, the meat and milk become contaminated with the toxin. When milk or meat containing the toxin is consumed, the poison is passed on to humans. If consumed in large enough quantities, it can cause tremetol poisoning in humans. The poisoning is also called milk sickness, as humans often ingested the toxin by drinking the milk of cows that had eaten snakeroot.[6]
Rosary Pea
Also called jequirity beans, these piously-named seeds contain abrin, an extremely deadly ribosome-inhibiting protein. Rosary peas are native to tropical areas and are often used in jewelry and prayer rosaries. While the seeds are not poisonous if intact, seeds that are scratched, broken, or chewed can be lethal. It only takes 3 micrograms of abrin to kill an adult, less than the amount of poison in one seed, and it is said that numerous jewelry makers have been made ill or died after accidentally pricking their fingers while working with the seeds. Like ricin, abrin prevents protein synthesis within cells and can cause organ failure within four days.
Abrus precatorius, commonly known as jequirity bean or rosary pea, is a herbaceous flowering plant in the bean family Fabaceae. It is a slender, perennial climber with long, pinnate-leafleted leaves that twines around trees, shrubs, and hedges.
The toxin abrin is a dimer consisting of two proteinsubunits, termed A and B. The B chain facilitates abrin's entry into a cell by bonding to certain transport proteins on cell membranes, which then transport the toxin into the cell. Once inside the cell, the A chain prevents protein synthesis by inactivating the 26S subunit of the ribosome. One molecule of abrin will inactivate up to 1,500 ribosomes per second.
Symptoms are identical to those of ricin, except abrinis more toxic by almost two orders of magnitude; the fatal dose of abrin is approximately 1/75th that of the fatal dose of ricin (though the abrin in ingested seeds may be absorbed much more slowly than the ricin in Ricinus communis even if the seeds are chewed and the coat penetrated, allowing time for successful rescue efforts in at least some cases.[8]) Abrin has an LD50 of only 0.56 μg/kg in mice, and Kingsbury lists a toxic dose in humans at 0.00015% body weight, or approximately 0.1 mg for a 150 lb human.[9] Ingesting intact seeds may result in no clinical findings, as they can pass undigested through the gastrointestinal tract because of their hard shell.[10]
This plant is also poisonous to horses.[11]
Symptoms of poisoning include nausea, vomiting, convulsions, liver failure, and death, usually after several days.
Death Cap
Perhaps the deadliest of all mushrooms, the death cap is found throughout Europe and closely resembles edible straw mushrooms and caesar's mushrooms. Its heat-stable amatoxins withstand cooking temperatures and quickly damage cells throughout the body. Within 6 to 12 hours after consumption, violent abdominal pain, vomiting, and bloody diarrhea appear, causing rapid loss of fluid from the tissues and intense thirst. Signs of severe involvement of the liver, kidneys, and central nervous system soon follow, including a decrease in urinary output and a lowering of blood sugar. This condition leads to coma and death in more than 50 percent of the incidents. Notable deaths include Pope Clement VII, who died of accidental death cap poisoning in 1534, and possibly Roman Emperor Claudius in 54 CE.
Amanita phalloides /æməˈnaɪtə fəˈlɔɪdiːz/, commonly known as the death cap, is a deadly poisonous basidiomycete fungus, one of many in the genus Amanita. Widely distributed across Europe, but now sprouting in other parts of the world, A. phalloidesforms ectomycorrhizas with various broadleaved trees. In some cases, the death cap has been introduced to new regions with the cultivation of non-native species of oak, chestnut, and pine. The large fruiting bodies (mushrooms) appear in summer and autumn; the caps are generally greenish in colour with a white stipe and gills. Cap colour is variable, including white forms (see Taxonomy below), and thus not a reliable identifier.
These toxic mushrooms resemble several edible species (most notably Caesar's mushroom and the straw mushroom) commonly consumed by humans, increasing the risk of accidental poisoning. Amatoxins, the class of toxins found in these mushrooms, are thermostable: they resist changes due to heat, so their toxic effects are not reduced by cooking.
A. phalloides is one of the most poisonous of all known mushrooms. It is estimated that as little as half a mushroom contains enough toxin to kill an adult human. It has been involved in the majority of human deaths from mushroom poisoning,[1] possibly including the deaths of Roman Emperor Claudius in AD 54 and Holy Roman Emperor Charles VI in 1740. It has been the subject of much research, and many of its biologically active agents have been isolated. The principal toxic constituent is α-amanitin, which damages the liver and kidneys, causing liver and kidney failure that can be fatal
