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Inert gas causes cerebral hypoxia and thus fading of awareness and eventual unconsciousness (which occurs ~30 mmHg) so rapidly that any dyspnea as a result of hypoxia is extremely minimal given a proper setup, especially if hypocapnia is induced beforehand through hyperventilation and if adequate CO2 purging is present either through exhalation valves or high flow rate flushing it out.
Yeah, maybe CO and inert gas (N2, Ar, He) but its difficult to achieve those concentrations of CO and guarantee that there isnt going to be leaks of gas. Also, for me, the consequences of doing it wrong are very scary (brain damage or being a vegetable)
I was wondering the same thing… There are plenty of videos of pilots going through high altitude training, where the o2 levels are gradually reduced in the room to simulate high altitude. You seem them start to lose coordination, get confused, and giggly as their o2 levels drop. Sometimes they take them almost to the point of passing out… and there doesn't seem to be any distress. So since inert gases would act the same way, displacing the o2, even with an improper, slower setup, there still shouldn't be much discomfort, correct?
I think that an improper, slow setup could lead to some distress (probably mainly nausea, headaches, things like that rather than air hunger, but potentially some if hypoxia is severe enough without knocking one out)
pmc.ncbi.nlm.nih.gov
And there was a feeling of suffocation, how was the person, was she breathing heavily? during the 15 minutes, what happened? I spoke with a person from an "association" who attended, from start to finish, several end-of-life experiences with sn (it's real, it's not fake), he told me that we lost consciousness quite quickly...afterwards it was only elderly people so surely less resistant and the result is perhaps distorted with younger, healthy people...
Send me a dm pls I would forward the experience to u
Sure u just have to be brave to accept that
I don't think so. More like no method most people can access won't be unpleasant. A proper inert gas should not be unpleasant, for example.
To reduce and eliminate SI, fear, panic and anxiety, the protocol provides for benzos, which are extremely effective in this sense. Without benzos, the whole process can be very frustrating.
Acidosis is certainly the main cause of Kussmaul's breathing, and as for hypocapnia, following the recorded cases on this forum, we could notice that almost everyone reported tingling of the hands or legs, which is one of its main symptoms. Essentially, the question of all questions in a practical sense for the implementation of the SN protocol is how to accelerate the loss of consciousness and reduce anxiety and fear. Currently, psychosedatives and tranquilizers are the only means of choice in this case, and with them, the timing of ingestion and dosage are key.AIR HUNGER: A PRIMAL SENSATION AND A PRIMARY ELEMENT OF DYSPNEA - PMC
The sensation that develops as a long breath hold continues is what this article is about. We term this sensation of an urge to breathe ‘air hunger’. Air hunger, a primal sensation, alerts us to a failure to meet an urgent homeostatic need ...
"There are fewer data relating air hunger ratings to levels of hypoxia. Hypoxia is a relatively weak stimulus, as might be expected from ventilatory response data"
"Hypoxia during free breathing is a notoriously poor stimulus for air hunger; the relief of air hunger produced by increased mechanoreceptor input and by the accompanying hypocapnia blunts air hunger to the point that subjects usually have severe decline in cognitive function or lose consciousness before reporting air hunger"
So, I think it's reasonable to conclude that air hunger from SN is probably caused almost exclusively by acidosis, especially considering that Kussmaul respirations caused by acidosis will induce hypocapnia.
yes but you still have to be sure that you are really unconscious during the grunts... and that it's not that you can't move...
For the cases I mentioned, it is quite certain that they were unconscious. One of the problems with benzo is that it causes resistance, you would have to completely "clean" yourself from benzo in order to achieve the desired effect with the appropriate shock dose.
Why do you think some people experience burning sensation in the throat/stomach and/or pain (headache?), but others do not? I heard someone propose that it's because SN reacts with stomach acid and that an antacid would help with this, but I'm unsure how true this is.
Unfortunately, we can only guess about it, but this thesis of yours regarding stomach acid seems quite rational and certain to me. In general, people react differently to medications, food and even poisons, it is extremely individual and represents a complex set of numerous factors (body condition, age, body weight, health, some genetically predisposed factors and so on). However, there are conflicting opinions about antacids, and according to my knowledge, they are sometimes recommended and sometimes excluded from the protocol. There are some theories according to which Antacids generally slow down the effects of SN and thus prolong the whole process, while others claim exactly the opposite. I think the latest protocol from 2024 does not recommend the use of Antacids (someone wrote about it here on the forum).
The latest protocol does not recommend them, yes, but I don't think we should put much stock in the PPH when there are so many errors and inconsistencies in it. I'll do more research on the physiology of them and SN ig.
I would think the burning sensation is caused by the saltiness and maybe some of them had ulceras/irritated esophagus from before.
What kind of inconsistencies yu have detected? No offense, Im justt really curious
That might be.
I suspect that some samples of NaNO2 may have traces of NaOH among the impurities, since one of the ways to produce NaNO2 implies using NaOH as one of the reagents
I'm just going to address the issues with their SN protocol, because I don't feel like typing out the rest I found.
Thank you for your input. I'll look into this.
Also meto only is mentioned as an anti emetic. But in the "best" method DDMPAh Meto + Ondensatron is mentioned as an anti emetic.
Yeah, I noticed that as well. Odd that they only advise this for DDMAPh.
I agree. I plan to combine meto and ondansetron for this exact reason.
100%. Nitschke and company are focused primarily on the sarco pod, and it shows
Yeah, pretty obvious that they are putting all the odds on Sarco, which I find sad because very few people can afford go to a country that have a Sarco or afford to print the 3d model that they want to make public, allegedly.
