Sn takes hours to kill someone, it just takes up to 20 minutes to pass out. In most cases like this it is cyanide. Sn takes four to five hours to kill, cyanide takes 5 to 20 minutes.
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Secrets1
Specialist
- Nov 18, 2019
- 364
Ok. What you're stating is far from conclusive for any given individual. Maybe I jumped the gun saying I'd bet the farm on SN. Maybe not…. We don't know all the details.
That's true. BUt in every previous case I have seen with this in the news it was cyanide, and there have been a few of these- one or two cases loike this on foresic files as well.
Secrets1
Specialist
- Nov 18, 2019
- 364
So do you think you know about every relevant case then?? Citing forensic files a tv show?
. We'll find out the truth shortlyForensic Files is a very reputable show which depicts true crimes- and their facts always check out. I've explained my reasoning about the time each method takes and the facts in previous cases- I just don't know why you think sn would work that fast. PLus the telltale blue skin would lead to an antidote being used in plenty of time to save him- so sn couldn't work for a public situation like this where someone needs something to work fast- something there is no antidote for.So do you think you know about every relevant case then?? Citing forensic files a tv show?
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Hollowillow
The only place that allows negative feelings.
- Aug 7, 2022
- 1,515
I've been abused. If he's guilty I have mixed feelings. Glad he's no longer there to hurt, sad he won't bend over in a shower & get justice, jealous af
Hi.
Where does it say he collapsed immediately?
Hi.
From the research I am gathering from numerous medical articles in relation to sodium nitrite. They are mentioning "ashen" and "ashen grey skin".
They also don't seem to mention that the skin turns blue as much as the lips, fingertips, and toes do.
Nobody knows what the suspect took.
No sense in trying to argue and prove someone wrong.
He could have taken Sodium Azide for all we know.
Hi.
Going by the observed cases from the "SN megathread" posting by a user from here, everyone died in under an hour from Sodium Nitrite toxicity.
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It seems everyone is speculating that SN was used. But perhaps it was SA? (Sodium Azide). I don't know, of course, just my 2 cents.
SA is an odorless, tasteless, water-soluble crystalline powder. Exposure can cause rapid breathing and heart rate, low blood pressure, loss of consciousness, and respiratory failure leading to death.
SA causes dizziness, tachycardia, and fainting within seconds after ingestion. Symptoms, including decreased mental status, dizziness, fainting, tingling, hypotension, and fainting, develop minutes after consuming SA. Acute toxicity following exposure to SA is manifested by nausea, vomiting (as mentioned in the article), headache, reduced blood pressure, tachycardia, cardiac muscle ischemia, and metabolic acidosis. The final stage of poisoning may be characterized by coma, bradycardia, convulsions, cardiorespiratory arrest, and, ultimately, death.
SA is primarily a mitochondrial toxin, which binds the electron transport chain, inhibiting oxidative phosphorylation. The resulting reduction in adenosine triphosphate (ATP) production, even in the presence of oxygen, results in metabolic failure. This mechanism of action is similar to that of cyanide, although SA causes more pronounced vasodilation due to the in vivo conversion of some azide to the vasodilator nitric oxide. Some reports suggest that azide lethality is due to enhanced excitatory transmission from nitric oxide in the central nervous system.
There is no specific antidote for SA intoxication. A total of 185 individual SA cases have been reported concluding a 100% success rate in suicide cases.
SA is an odorless, tasteless, water-soluble crystalline powder. Exposure can cause rapid breathing and heart rate, low blood pressure, loss of consciousness, and respiratory failure leading to death.
SA causes dizziness, tachycardia, and fainting within seconds after ingestion. Symptoms, including decreased mental status, dizziness, fainting, tingling, hypotension, and fainting, develop minutes after consuming SA. Acute toxicity following exposure to SA is manifested by nausea, vomiting (as mentioned in the article), headache, reduced blood pressure, tachycardia, cardiac muscle ischemia, and metabolic acidosis. The final stage of poisoning may be characterized by coma, bradycardia, convulsions, cardiorespiratory arrest, and, ultimately, death.
SA is primarily a mitochondrial toxin, which binds the electron transport chain, inhibiting oxidative phosphorylation. The resulting reduction in adenosine triphosphate (ATP) production, even in the presence of oxygen, results in metabolic failure. This mechanism of action is similar to that of cyanide, although SA causes more pronounced vasodilation due to the in vivo conversion of some azide to the vasodilator nitric oxide. Some reports suggest that azide lethality is due to enhanced excitatory transmission from nitric oxide in the central nervous system.
There is no specific antidote for SA intoxication. A total of 185 individual SA cases have been reported concluding a 100% success rate in suicide cases.
