
pthnrdnojvsc
Extreme Pain is much worse than people know
- Aug 12, 2019
- 3,418
Your post is full of bad assumptions and condescension . SN hasn't caused brain damage in all the cases i've seen because in hypoxia SN dilates the blood vessels . You are spreading misinformation.Your post is full of bad assumptions. Extreme vasodilation in the brain is capable of producing a stroke as is vasoconstriction. SN isn't neuroprotective.
I haven't seen a single reliable case of brain damage from an SN ctb attempt survivor : and i'v seen several here including 16 documented in that post with SN cases. So SN is neuroprotective. That you say it's not is spreading misinformation .Furthermore you do it in a derogatory and condescending manner which violates the rules of this forum
These are quotes from the following article :
" Nitrite is a major intravascular store for nitric oxide. The conversion of nitrite to the active nitric oxide occurs mainly under hypoxic conditions to increase blood flow where it is needed the most "
"...intravenous sodium nitrite reduced the damage to the heart [24], liver [25] and brain after ischaemia/reperfusion [26], prevented [27] and reversed [28] cerebral vasospasm after subarachnoid haemorrhage (SAH)2 in non-human primates. "

Effect of nitrite on the electroencephalographic activity in the healthy brain
Nitrite is a major intravascular store for nitric oxide. The conversion of nitrite to the active nitric oxide occurs mainly under hypoxic conditions t…
But for ctb puposses people take many times that amount.This proves that in low dose of SN:

Low-dose sodium nitrite vasodilates hypoxic human pulmonary vasculature by a means that is not dependent on a simultaneous elevation in plasma nitrite - PubMed
Inorganic nitrite has recently been recognized to possess vascular activity that is enhanced in hypoxia. This has been demonstrated in humans in the forearm vascular bed. In animal models nitrite reduces pulmonary vascular resistance, but its effects upon the pulmonary circulation of humans have...


Sodium nitrite exerts an antihypertensive effect and improves endothelial function through activation of eNOS in the SHR - Scientific Reports
Sodium nitrite (NaNO2) induces relaxation in isolated arteries partly through an endothelium-dependent mechanism involving NO-eNOS-sGC-cGMP pathway. The present study was designed to investigate the effect of chronic NaNO2 administration on arterial systolic blood pressure (SBP) and vascular...

https://sanctioned-suicide.net/thre...nd-experiences-information-google-docs.29900/
Why don't you tell then the reason for no brain damage -in all of the survivors documented on that thread for SN cases ,then if it is not vasodilation of the brain blood vessels in hypoxia. You don't provide a counter reason.
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